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by Anthony Roberts - Testosterone Suspension
does not have an ester attached, making it a "pure"
version of the parent hormone, Testosterone. Therefore,
there is no ester to be accounted for in the weight.
This means 100 mg of testosterone suspension contains
100 mg of the steroid. This makes it the most potent
forms of Testosterone available. Gains come quicker
and side effects much more possible with such a
strong version of this hormone.
Because it does not need to break down an attached
ester (like other esterified versions) the hormone
is effective immediately when administered. The
users testosterone levels will be raised for only
2-3 days (at most) after injection and the steroid
should be administered daily. This testosterone
is dissolved in water typically, not oil like most
of the other esterified versions making it more
readily available in the body.
Background
Testosterone Suspension was the first anabolic
steroid ever developed, in the 1930’s. It has been
used as the most potent mass builder for decades.
It was responsible for the Russian dominance in
weightlifting not long after.
In the United States, a very short acting version
of Testosterone Suspension is available. The brand
name for aqueous testosterone suspension is "Aquaviron."
There are only a few overseas versions available,
including, the most popular, Testosus, which is
manufactured in Australia. Testosus was recently
replaced by a Mexican brand known as Anabolic-TS,
which is the same steroid now sold under the label
Grupo Comercial Tarasco (a Mexican distributor for
Jurox). Univet Uni-test is the Canadian version
of Testosterone Suspension. Many underground labs
also suspend this variance of Testosterone (which
is usually suspended in water) in propylene glycol
or oil, making it a much more painful injection.
Steroid Action
The key role of Testosterone include: promoting
health and well-being through enhanced libido, energy,
immunity, increased fat loss, gaining and maintaining
lean muscle mass, preventing Osteoporosis (loss
of bone density) and possible protection against
heart disease. Testosterone is also responsible
for normal growth and development of male sex organs
and maintenance of secondary sex characteristics.
Secondary sex characteristics are specific traits
that separate the two sexes, but are not directly
part of the reproductive system, for example: chest
and facial hair, a distinguished jaw line, broad
shoulders and increased muscle mass. Testosterone
binds to the Androgen Receptors (AR), which thus
causes accelerated muscle gain, fat loss, and muscle
repair and growth. These mechanisms are stimulated
by activation of the Androgen Receptors (either
directly or as DHT)
Testosterone Suspension is the best-known potential
mass builder, yet must be used with caution due
its high risk of side effects. Often these effects
are the result of the hormone being so readily converted
with in the human body to a more androgenic form
of itself, Dihydrotestosterone (DHT). Through aromatization,
testosterone can also in-turn form estrogen (the
female sex hormone). These potential side effects
include: water retention, bloating, fat gain, gynecomastia
(formation of breast tissue in males) and conversion
to DHT (dihydrotestosterone) as well. Also, supplementing
Testosterone will result in the shutting down of
the body's natural production of the hormone. The
severity of these effects depends mostly on the
levels and duration of circulating free testosterone.
Testosterone's anabolic/androgenic effects are dependant
upon dosage, the higher the dose the higher the
muscle building effect. Testosterone promotes aggressive
and dominant behavior. Effective doses range from
350-1000 mg per week (50-140 mg/day).
Technical Data
Users of Testosterone Suspension reported rapid
muscle growth and water retention. Increased aggression
was also commonly reported with use of this specific
type of Testosterone. Users found that Testosterone
Suspension provided the best results when it was
run for at least 8 weeks. More experienced users
found they had better results when extending their
cycle slightly longer (1). While using this hormone,
endurance and strength increases were reported while
following an intense training and workout routine
(1-2).
As with all of the Testosterones, the most common
report by subjects using testosterone was immense
gains in strength (3). Alterations in size, shape,
and appearance of the muscle were also reported
(4). There was roughly a 15% gain in Lean Body Mass
from 20 weeks of 600 mg/week of Testosterone therapy.
(5)
User Notes
Most of the preceding information on testosterone
suspension was actually about testosterone in general
or other esters. Why? Well, medically, there is
nobody using testosterone suspension anymore. It’s
considered a bit of an ugly drug by most of the
medical community. It’s just straight testosterone
suspended in water (or sometimes, with certain underground
preparations in oil). As a result, it needs to be
injected at least once a day, with some athletes
preferring to inject it twice a day.
It has a reputation for being used as a pre-game
(or pre-workout) stimulant to increase aggression.
I can attest to it being pretty useful for this
purpose, actually, as can many pre-contest bodybuilders.
In the end, though, not many people will base
a cycle around testosterone suspension - it’s uncomfortable
to inject and leaves some pretty odd looking bumps
in the muscle while it’s still there.
Testosterone Suspension Resources
Tesosterone Suspension Pictures
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Trivial name
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Testosterone [USAN:INN]
(base)
|
| Chemical name |
17ß-hydroxyandrost-4-en-3-one
|
| Systematic Name |
(8S,9S,10R,13S,14S,17S)-17-hydroxy-10,13-dimethyl-1,2,6,7,8,9,11,12,14,15,16,17-dodecahydrocyclopenta[a]phenanthren-3-one
|
| Index name |
Androst-4-en-17beta-ol-3-one |
| CAS number |
58-22-0 |
| Empirical formula |
C19-H28-O2 |
| Merck Index Number |
Merck 11, 9109 |
| Molecular weight |
288.424 g/mol |
| Pregnancy category |
X |
| Legal status |
Prescription
only (US); DEA Schedule III (US) |
| Routes of administration |
Transdermal |
References
- Zhonghua Nan Ke Xue. 2003;9(4):248-51
- J Clin Endocrinol Metab. 2004 Oct;89(10):5245-55
- J Lab Clin Med. 1995 Mar;125(3):326-33.
- Am J Physiol. 1998 Nov;275(5 Pt 1):E864-712
- Am J Physiol Endocrinol Metab. 2001 Dec;281(6):E1172-81.
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